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One study found elevated serum ferritin levels and increased transferrin saturation in people awards with NASH. Hepatic iron index and hepatic iron levels are usually in the normal range (Younossi et al, 1999), but studies that looked at the possibility of hepatic iron overload as an aetiological factor have shown that hepatic iron deposition and the HFE gene of familial haemochromatosis are not necessary for the development of NAFLD (Bugianesi et al, 2004). Clinical diagnosis of NAFLD NAFLD should be suspected in a patient with awards liver disease in whom awards other common causes, such as alcohol, viral infection, drugs, and autoimmune, genetic or metabolic disorders have been excluded. Careful exclusion of alcohol as an aetiological factor is required, and it has been suggested that a cut-off level of 14-28 units per week is needed to differentiate between alcohol-related and NAFLD (Brent et al, 2003).
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